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Hyperinflammation and airway surface liquid dehydration in cystic fibrosis: purinergic system as therapeutic target

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Following the initial generation, most of the pro-BDNF is then packaged into vesicles in a regulated pathway and undergoes N-terminal cleavage by extracellular proteases, such as plasmin, metalloproteinase gene matrilysin (MMP7) (7,8), tPA/plasmin cascade (8) and extracellular matrix-metalloproteinases (9)

Conversely, high oxidative stress can accelerate NAD+ degradation via the enzyme CD38, which consumes NAD+ as part of the inflammatory response