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A marked reduction in the expression levels of ferroptosis-related regulatory proteins, particularly GPX4 and FTH1, was distinctly observed ( The implications of the study have identified ferroptosis as a viable pathway for therapeutic intervention in the context of RM, substantiating the assertion that the modified Shoutai Pill effectively mitigates this particular mode of cellular demise

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Notably, it can stimulate the differentiation of bone marrow mesenchymal stem cells into osteoblasts (the cells responsible for building new bone)