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Each Schwann cell modifies its basal lamina as to accommodate the regrowing axons and, at the same time, clears myelin debris and modulates the composition of the surrounding endoneurium for regrowing axons
doi: 10.1016/j.aca.2012.12.054
SETDB1 deficiency clearly promoted the acquisition of the TGF--induced mesenchymal phenotype but reduced the expression of E-cadherin in human alveolar epithelial cells by catalyzing the deposition of H3K9me3 on Snai1 , which inhibited the protein expression of Snai1, the main transcription factor that initiates the epithelial-mesenchymal transition (EMT)
& Kitchen, D
