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Further supporting a pathobiological role for Abeta in PD, alpha-synuclein and amyloid beta double transgenic mice showed more Lewy Body pathology than the alpha synuclein gene mutation alone [73]
U2OS cells with stably overexpressing Flag-GCLC were treated with TBH or H 2 O 2
It was further discovered that a higher ascorbate-glutathione pool protects against oxidative damage in the arsenate-tolerant As hyperaccumulator P
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