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STAT3 as a newly emerging target in colorectal cancer therapy: tumorigenesis, therapy response, and pharmacological/nanoplatform strategies

Preclinical and early-stage research literature has explored GHK-Cu in relation to oxidative stress response systems, inflammation-associated signaling pathways, and gene expression patterns linked to tissue maintenance

Interestingly, it is found that GSTP1 expression is regulated by the Keap1-Nrf2-ARE pathway and GSTP1 catalyzes the S-glutathionylation of cysteine residues of Keap1 protein, while S-glutathionylation of Keap1 leads to Nrf2 activation and increases the expression of GSTP1 (Ikeda et al., 2002