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This unique substrate specificity and acid stability make RmuAP1 a promising candidate for oral enzyme therapy to aid gluten digestion in individuals with CD, potentially reducing gluten-induced intestinal damage (Zhang et al

Reduced ER expression and impaired ER function have been linked with increased prevalence of certain aspects of the metabolic syndrome in both male and female humans and rodents (137141), and certain polymorphisms of the human ER gene are associated with abnormal adiposity (138, 142)

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The effect of inosine in adipocytes was shown to be exerted through the activation of PKA which in turn enhances fat metabolism via increased UCP-1 activity