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Even though some studies have shown that alveolar type II cells can supplement endogenous synthesis of GSH with the uptake of exogenous GSH (11, 12), the antioxidant effectiveness of exogenously administered GSH is hindered by its instability when crossing cell membranes and its rapid hydrolysis in the circulation (13-15)

Activation of human B cells mediated through two distinct cell surface differentiation antigens, Bp35 and Bp50
