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Cardiac hypertrophy has been inversely correlated with the expression of miR-1 (Sayed et al., 2007) and miR-1 depleted mice show cardiac defects, including misregulation of cardiac morphogenesis, electric conduction, and cell proliferation (Yang et al., 2007
One emerging therapeutic strategy involves inhibition of NADPH oxidase (NOX) enzymatic activity, with compounds such as ebselen, S17834, and GKT137831 showing potential across various disease models
Retinoids, LRAT and REH activities in eggs of Japanese quail following maternal and in ovo exposures to 3, 3, 4, 4-tetrachlorobiphenyl
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