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However, a deeper comprehension of the DA catabolic pathway and how it functions in PD patients will enable the development of more individualized and efficient therapy approaches

49,72 Dysregulation of this feedback contributes to chronic inflammation, fibrosis, and tumorigenesis, and restoring this balance represents a promising therapeutic strategy

The possible mechanism is that glucose metabolism disorder and Hcy metabolism worsen each other, resulting in the increase of Hcy level (51)

Drugs used as MAO-A inhibitors were evaluated as a reference comparison
