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Similarly, histone deacetylase (HDAC) inhibitors such as trichostatin A or entinostat may reverse the transcriptionally repressive states established in DTP cells, restoring sensitivity to initial anticancer treatments (Sharma et al., 2010

The nanoparticle-loaded edaravone showed reduced edema as compared to free edaravone treated group, significantly improved neurological function, and reduced the production and release of interleukin and tumor necrosis factor, which was considered beneficial to protect healthy brain tissue and other organs, and conducive to the recovery and healing [275]