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Previous research has shown that GPX4 plays a reno-protective role in tubular cells, with GPX4-null mice exhibiting marked increases in tubular cell death and proteinuria (12)

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also referred to as GSHS in some literature, EC 6.3.2.3.), in which glycine is added to -glutamylcysteine to form glutathione, takes place in plastids and the cytosol as GSH2 is targeted to both chloroplasts and the cytosol (Noctor et al., 2002

(42,115) Unfortunately, the absolute potential of the SHE and thus also the proton solvation energy varies randomly upon changing the basis set or electronic structure method, and no clear trends exist which could be exploited to systematically improve the accuracy of the direct p K a scheme