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MYC-driven mitochondrial metabolic rewiring increases DLAT expression and cuproptosis sensitivity in group-3 medulloblastomas, whereas DLAT inhibition lowers TCA cycle metabolism and GSH synthesis 175

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Therefore, it is crucial that treatment be aggressive to correct deficiencies along with lifelong screening to prevent bone loss, irreversible damage, and preventable complications

Studies also document TB-500's effects on basic fibroblast growth factor and vascular endothelial growth factor expression, both involved in blood vessel formation processes