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Use of acivicin in the determination of rate constants for turnover of rat renal -glutamyltranspeptidase

Moreover, since GSTs are heterogeneous enzymes with significant overlapping substrate specificities, the findings suggest that endogenous levels of other phase II enzymes cannot compensate adequately for the loss of GSTM1, and thus their induction by a pharmacologic/dietary factor such as sulforaphane may be required to lower oxidative stress in a disease state such as hypertensive kidney disease (Figure 1)

This includes proper institutional oversight, appropriate experimental design and compliance with laboratory safety standards for research compound use