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Oxidation of the ketone bodies provides the brain with an alternative fuel source to generate acetyl-CoA used for maintaining the mitochondrial pools of tricarboxylic acid cycle intermediates and production of reducing equivalents for subsequent ATP production through the mitochondrial respiratory chain, thus restoring the energy deficit characteristic of pyruvate dehydrogenase complex deficiency

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In contrast, bacterial lipopolysaccaride (LPS) is able to trigger thyroiditis in NOD H2h4 mice, by interacting with TLR4 expressed on thyrocytes