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M.FujitaM
When NETs are generated, S100A8/A9 can be released from the cytoplasm of neutrophils, activating surrounding immune cells and thus amplifying the inflammatory response (106)
In the context of diabetic myocardium with I/R injury, apelin overexpression exerts cardioprotective effects via suppressing oxidative stress and apoptosis through the Phosphoinositide 3-kinase (PI3K) and p38-MAPK signaling pathways in rats [158]
Conversely, the UGT1A rs8330 polymorphism has been associated with increased levels of paracetamol glucuronidation
