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Human salivary histatin 1 regulating IP3R1/GRP75/VDAC1 mediated mitochondrial-associated endoplasmic reticulum membranes (MAMs) inhibits cell senescence for diabetic wound repair

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Instead, under these lactate-rich conditions, the highly expressed NAMPT utilizes an alternative signaling capacity to enhance immunosuppressive phenotype of TAMs through the HIF-1/STAT3 signaling pathway 23 , while preventing the utilization of salvaged NAD + for the glycolytic demands of an IFN-mediated inflammatory response 85