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Oxidative stress also contributes to thyrocyte apoptosis, with reduced antioxidant defenses (e.g., glutathione peroxidase and superoxide dismutase activity) often observed in patients with autoimmune thyroiditis
Phospho-RTK array analysis in lung cancer models identified increased IGF-1R phosphorylation in erlotinib-resistant adenocarcinoma cells with wild-type EGFR [106]
