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Damage-associated molecular patterns in myocardial infarction and heart transplantation: the road to translational success

In addition, increased antibody-dependent cellular phagocytosis was observed in Raji B cells or Jurkat T cells due to the presence of anti-CD38 or anti-CD3 autoantibodies (36)

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Various factors, including hyperglycemia, lipid accumulation, oxidative stress, hypoxia, RAAS, ER stress, inflammation, EMT and programmed cell death, have been shown to induce renal tubular injury and contribute to the progression of DKD
