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Another potential mechanism for age-dependent loss of antioxidant genes was provided by one of the studies showing that the presence of transcription repressor BACH1 along with the absence of coactivator CREB-binding protein (CBP) in the aged mice may convert NRF2 binding from an active ARE to an alternative ARE [114]

These neuropeptides can affect either the response of cortical cells in zona fasciculate to ACTH, the vascularization of cortex, its growth, or they may exert direct modulatory effects on the cortical cells themselves