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Mitochondrial metabolism and neuroinflammation Mitochondrial dysfunction acts as a core driver of ferroptosis through a combination of iron homeostasis imbalance and oxidative stress (59)

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Changes in genetics encompass MET exon 14 skipping mutations (METex14), amplification of MET genes, increased c-MET protein levels, structural domain mutations in MET kinase, and fusion of MET genes [22]

A frozen vial may look identical to an unfrozen one after thawing
