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But the jump from C57BL/6 mice to humans involves differences in metabolic rate, NNMT tissue distribution, adipose tissue physiology, and drug metabolism that preclinical data cannot address
Moreover, adipocytes are reported to modulate responses to treatment
I would suggest that you need to read all of the studies before you reach a conclusion
Associated with elevated NAD+ levels and SIRT1 activation in preclinical studies [2] [3]
