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[DOI] [PubMed] [Google Scholar] 40.Sharpe M.C., Archard L.C., Banatvala J.E., Borysiewicz L.K., Clare A.W., David A., Edwards R.H., Hawton K.E., Lambert H.P., Lane R.J., et al
They demonstrated that mutant form of PLC that lost its PIP2 hydrolyzing properties still can activate Ras-MAPK kinase pathway suggesting that this activation is due to Ras binding and GEF domain rather than to PIP2 hydrolysis [5]
