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This study not only elucidates the mechanisms of PARP1-mediated PXR E194 ribosylation in AILI but also identifies Sch B as the first specific inhibitor of this pathway, providing a theoretical basis for precision detoxification strategies targeting posttranslational modifications.

As a precursor that supports the production of one of the body’s key antioxidant compounds, glutathione, N-acetyl-L-cysteine (NAC) is well-positioned in the cellular defense arena

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