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Hammi Meriam, for their invaluable guidance and unwavering support throughout this research
Cao D, Liu Y, Cheng Y, et al
This situation was first reported in a boy with a deletion of exon 4 of the IGF-I gene (90) resulting in severe fetal and post-natal growth arrest, poor brain development and extreme insulin resistance with compensatory hyperinsulinemia and acanthosis nigricans
