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and releasing glutamate, quinolinic, and arachidonic acid, which may all contribute to neuron damage [131]
Phillyrin relieves lipopolysaccharide-induced AKI by protecting against glycocalyx damage and inhibiting inflammatory responses
The regulatory role of HDAC6 in podocytes autophagy and motility indicates that HDAC6 functions as a DKD treatment target (Liang et al., 2020)
Preclinical models demonstrate that such engineered oncolytic viruses can reshape the tumor microenvironment (TME) towards an immune-activated state, thereby synergizing with ICIs to improve therapeutic efficacy (203)
