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Mutagenesis of the active site of the human Theta-class glutathione transferase GSTT22: catalysis with different substrates involves different residues
A urate gene-by-diuretic interaction and gout risk in participants with hypertension: results from the ARIC study
The oxidative stress in mitochondria could be counteracted by IDH1-driven reductive carboxylation in the cytosol, followed by the transfer of citrate into the mitochondria, which was to be oxidized by IDH2 to generate NADPH [104]
Moreover, SLC7A5 inhibitors could also be used for the treatment of patients with NSCLC without EGFR mutations (93)
