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Longitudinal studies in animal models and post-mortem human tissues are needed to determine whether microglial iron overload is a cause or consequence of -syn pathology, and to identify the disease stage at which glial modulation is most therapeutically beneficial
50 AT-II has a pivotal role not only in the progression of tubulointerstitial injury but also in obstructive nephropathy
Mitochondrion 49 , 269283 (2019)
