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Novel aspects of toxicity mechanisms of dioxins and related compounds
For example, low concentrations of HO promote cell survival by inhibiting the phosphatase PTEN and activating the PI3K/Akt pathway, while superoxide anions (O) produced by NOX 2 in macrophages induce pro-inflammatory cytokine expression via NF-B signaling to enhance antimicrobial immunity 18,19,20
The coordinated regulation of multiple signaling pathways generates synergistic protective effects, addressing the multifactorial nature of reproductive oxidative stress through the engagement of diverse cellular defense systems
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